School personnel information

写真b

NODA Takeshi


Keyword

Autophagy lysosome vacuole protein degradation Infectious Diseases yeast

Mail Address

Mail Address

Laboratory Phone number

+81-6-6879-2976

Laboratory Fax number

+81-6-6879-2110

URL

http://www.researcherid.com/rid/B-7240-2008

Gender

Male

Organization 【 display / non-display

  • 2006.02.01 - 2007.03.31, Research Institute for Microbial Diseases, Associate Professor, full time

  • 2007.04.01 - 2010.03.31, Research Institute for Microbial Diseases, Associate Professor, full time

  • 2010.04.01 - 2012.04.30, Department of Frontier Biosciences, Graduate School of Frontier Biosciences, Associate Professor, full time

  • 2012.05.01 - , Center for Frontier Oral Science, Graduate School of Dentistry, Professor, full time

  • 2010.04.01 - 2012.04.30, Division of Medicine, Graduate School of Medicine, Associate professor, part time

  • 2012.06.01 - , Graduate School of Frontier Biosciences, Professor, part time

Education 【 display / non-display

The University of Tokyo Faculty of Liberal Arts  Graduated 1991.03
The University of Tokyo Graduate School, Division of Science  Completed 1993.03
The University of Tokyo Graduate School, Division of General Culture  Completed Ph.D. 1996.03

Employment Record 【 display / non-display

National Institute for Basic Biology Assistant Professor 1996.04 - 2006.01
University of California San Diego, VIsiting Scientist 2003.05 - 2005.11

Research topics 【 display / non-display

  • Cell biology
    Microbiology
    Infection
    Cell biology, Functional basic dentistry, Living organism molecular science

 

Academic Papers 【 display / non-display

  • Three-Axis Model for Atg Recruitment in Autophagy against Salmonella., Noda T, Kageyama S, Fujita N, Yoshimori T., Int J Cell Biol., 2012:389562. 1-6, 2012.02, Review Papers

  • Dysfunction of autophagy participates in vacuole formation and cell death in cells replicating hepatitis C virus., Taguwa S, Kambara H, Fujita N, Noda T, Yoshimori T, Koike K, Moriishi K, Matsuura Y*., J Virol., 85(24), 13185-13194, 2011.12, Papers

  • Atg14L recruits PtdIns 3-kinase to the ER for autophagosome formation., Noda T, Matsunaga K, Yoshimori T., Autophagy., 7,1-2, 2011.04, Review Papers

  • The LC3 recruitment mechanism is separate from Atg9L1-dependent membrane formation in the autophagic response against Salmonella, Shun Kageyama, Hiroko Omori, Tatsuya Saitoh, Takefumi Sone, Jun-Lin Guan, Shizuo Akira, Fumio Imamoto, Takeshi Noda, Tamotsu Yoshimori, Molecular Biology of the Cell, 22, 13, 2290-2300, 2011.04, Papers

  • Chemical modulators of autophagy as biological probes and potential therapeutics., Fleming A, Noda T, Yoshimori T, Rubinsztein DC., Nat Chem Biol., 7, 9-17, 2011.01, Review Papers

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Books 【 display / non-display

  • Special, Monitoring autophagy in mammalian cultured cells through the dynamics of LC3. Methods Enzymol., Kimura S, Fujita N, Noda T, Yoshimori T., Academic Press,Inc., 2009.02

  • Special, Macroautophagy in Yeast in "AUTOPHAGY", Noda T. and Ohsumi Y, LANDES Bioscience, 2003.01