School personnel information


NODA Kanae

Organization 【 display / non-display

  • 2008.07.01 - 2010.03.31, Research Institute for Microbial Diseases, Specially Appointed Researcher

  • 2010.04.01 - 2011.03.31, Division of Preventive and Environmental Medicine, Graduate School of Medicine, Specially Appointed Researcher

  • 2011.04.01 - 2012.04.30, Division of Medicine, Graduate School of Medicine, Specially Appointed Researcher

  • 2019.04.01 - , Center for Frontier Oral Science, Graduate School of Dentistry, Specially Appointed Researcher


Academic Papers 【 display / non-display

  • The PtdIns3-phosphatase MTMR3 interacts with mTORC1 and suppresses its activity., Hao F, Itoh T, Morita E, Shirahama-Noda K, Yoshimori T, Noda T, FEBS letters,590(1) 161-73, 2016.01, Papers

  • Reciprocal conversion of Gtr1 and Gtr2 nucleotide-binding states by Npr2-Npr3 inactivates TORC1 and induces autophagy., Shintaro Kira, Keisuke Tabata, Kanae Shirahama-Noda, Akiko Nozoe, Tamotsu Yoshimori, Takeshi Noda, Autophagy,10(9):1565-78, 2014.09, Papers

  • TRAPPIII is responsible for the vesicular transport from early endosomes to the Golgi apparatus that facilitates Atg9 cycling in autophagy., Shirahama-Noda K, Kira S, Yoshimori T, Noda T., J Cell Sci,126, 4963-4973, 2013.08, Papers

  • Two Beclin-1 binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages. , Matsunaga K, Saitoh T, Tabata K, Omori H, Satoh T, Kurotori N, Maejima I, Shirahama-Noda, K, Ichimura I, Isobe T, Akira S, Noda T, Yoshimori T., Nature Cell Biol.,11(4), 385-396, 2009.04,, Papers