School personnel information

写真b

KITABATAKE Yasuji


Keyword

neuroscience, stem cell biology, pediatrics, iPS cell, Down syndrome

Mail Address

Mail Address

Laboratory Phone number

+81-6-6879-3932

Laboratory Fax number

+81-6-6879-3939

Gender

Male

Organization 【 display / non-display

  • 2009.11.01 - 2010.03.31, Graduate School of Medicine, Assistant Professor

  • 2010.04.01 - 2011.04.30, Osaka University Hospital, Assistant Professor

  • 2011.05.01 - 2017.02.28, Graduate School of Medicine, Assistant Professor

  • 2017.03.01 - , Osaka University Hospital, Associate Professor

Education 【 display / non-display

Osaka University Faculty of Medicine  Graduated 1995.03
Kyoto University Graduate School, Division of Medicine  Completed 2004.03

Research topics 【 display / non-display

  • Embryonic medicine and pediatrics-related, Cell biology-related, Neuroscience-general-related

  • Embryonic medicine and pediatrics-related

  • Embryonic medicine and pediatrics-related, Cell biology-related, Molecular biology-related, Embryonic medicine and pediatrics-related

  • Pediatrics
    Embryonic medicine and pediatrics-related

  • Embryonic/Neonatal medicine
    Embryonic medicine and pediatrics-related

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Academic Society Membership 【 display / non-display

  • JAPAN SOCIETY OF PERINATAL AND NEONATAL MEDICINE

  • JAPAN SOCIETY FOR NEONATAL HEALTH AND DEVELOPMENT

  • THE JAPANESE SOCIETY FOR REGENERATIVE MEDICINE

  • Trisomy 21 Research Society

 

Academic Papers 【 display / non-display

  • Drug development for Down syndrome, KITABATAKE YASUJI,2(13) 30-33, 2019.12, Other(In Japanese)

  • Elimination of protein aggregates prevents premature senescence in human trisomy 21 fibroblasts., Nawa N, Hirata K, Kawatani K, Nambara T, Omori S, Banno K, Kokubu C, Takeda J, Nishimura K, Ohtaka M, Nakanishi M, Okuzaki D, Taniguchi H, Arahori H, Wada K, *Kitabatake Y (* corresponding author), Ozono K, PloS one,14(7) e0219592, 2019.07, Other

  • Genetic correction of induced pluripotent stem cells mediated by transcription activator-like effector nucleases targeting ALPL recovers enzyme activity and calcification in vitro., Nakano C, Kitabatake Y, Takeyari S, Ohata Y, Kubota T, Taketani K, Kogo M, Ozono K, Molecular genetics and metabolism,127(2) 158-165, 2019.06, Other

  • Successful series of therapeutic approach in a case of giant omphalocele with diaphragmatic eventration and congenital diaphragmatic hernia,54(4) 1088-1093, 2018.12, Papers(In Japanese)

  • Systematic cellular disease models reveal synergistic interactions of trisomy 21 and GATA1 mutations in hematopoietic abnormalities, Banno K, Omori S, Hirata K, Nawa N, Nakagawa N, Nishimura K, Ohtaka M, Nakanishi M, Sakuma T, Yamamoto Y, Toki T, Ito E, Yamamoto T, Kokubu C, Takeda J, Taniguchi H, Arahori H, Wada K, Kitabatake Y, and Ozono K, Cell Reports,15 1-14, 2016.05, http://www.cell.com/cell-reports/abstract/S2211-1247(16)30444-2?_returnURL=http%3A%2F%2Flinkinghub.elsevier.com%2Fretrieve%2Fpii%2FS2211124716304442%3Fshowall%3Dtrue, Papers

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Lecture / Exhibition 【 display / non-display

  • 3rd International Conference of Trisomy 21 Research Society, Aneuploidy-associated stress; another therapeutic target for pathophysiology in Down syndrome, 2019.06

  • The 41st Annual Meeting of the Molecular Biology Society of Japan, A new concept of pathophysiology in Down syndrome, and new directions for drug discovery, 2018.11

  • The 41st Annual Meeting of the Molecular Biology Society of Japan, A new concept of pathophysiology in Down syndrome, and new directions for drug discovery, 2018.11

  • 2nd International Conference of the T21RS, A Pair of Maternal Chromosomes Derived from Meiotic Nondisjunction in Trisomy 21 Affects Nuclear Architecture and Transcriptional Regulation, 2017.06

  • 2nd International Conference of the T21RS, A Pair of Maternal Chromosomes Derived from Meiotic Nondisjunction in Trisomy 21 Affects Nuclear Architecture and Transcriptional Regulation, 2017.06