Organization 【 display / non-display 】
-
2004.10.01 - 2005.03.31, Research Institute for Microbial Diseases, Assistant Professor, full time
-
2005.04.01 - 2005.07.31, Research Institute for Microbial Diseases, Assistant Professor, full time
-
2005.08.01 - 2007.03.31, Research Institute for Microbial Diseases, Associate Professor, full time
-
2007.04.01 - 2011.06.30, Research Institute for Microbial Diseases, Associate Professor, full time
-
2011.07.01 - , Research Institute for Microbial Diseases, Specially Appointed Professor, full time
Research topics 【 display / non-display 】
-
Interaction of bacterial toxins with host cells
Cell biology, Bacteriology (including mycology)
Academic Papers 【 display / non-display 】
-
Crystallization and preliminary crystallographic studies of the HA3 subcomponent of the type B botulinum neurotoxin complex., Nishimura K, Kitadokoro K, Takegahara Y, Sugawara Y, Matsumura T, Karatani H, Fujinaga Y. , Acta Crystallogr Sect F Struct Biol Cryst Commun. , 67(Pt 10):1244-6, 2011.07, Papers
-
The botulinum toxin complex meets E-cadherin on the way to its destination., Sugawara Y, Fujinaga Y, Cell Adh Migr., 8;5(1), 2011.01, Review Papers
-
Interaction of botulinum toxin with the epithelial barrier., Fujinaga Y, Journal of Biomedicine and Biotechnology, Volume 2010, Article ID 974943, 9 pages doi:10.1155/2010/974943, Epub 2010 Feb 14, 2010.02, Review Papers
-
Botulinum HA disrupts the intercellular epithelial barrier by directly binding E-cadherin., Sugawara Y., Matsumura T, Takegahara, Y., Jin Y., Tsukasaki Y., Takeichi M., Fujinaga, Y, J. Cell Biol., 189:691-700, 2010.01, Papers
-
Disruption of the epithelial barrier by botulinum hemagglutinin (HA) proteins - Differences in cell tropism and the mechanism of action between HA proteins of types A or B, and HA proteins of type C., Jin Y1, Takegahara Y1, Sugawara Y, Matsumura T, and Fujinaga Y, Microbiology, 155: 35-45, 2009.01, Papers