School personnel information


Organization 【 display / non-display

  • 2007.04.01 - 2011.09.30, Institute for Protein Research, Professor

  • 2011.10.01 - , Research Institute for Microbial Diseases, Professor

Education 【 display / non-display

The University of Tokyo Faculty of Science  Graduated 1993.03
The University of Tokyo Graduate School, Division of Science  Completed 1995.03
The University of Tokyo Graduate School, Division of Science  Completed 1998.03

Employment Record 【 display / non-display

Research Associate, University of Tokyo 1998.04 - 2002.01
Associate Professor, University of Tokyo 2002.02 - 2007.03
Professor, Osaka University 2007.04 -

Research topics 【 display / non-display

  • Cell Biology


Academic Papers 【 display / non-display

  • The Oncogenic PRL Protein Causes Acid Addiction of Cells by Stimulating Lysosomal Exocytosis., Funato Y, Yoshida A, Hirata Y, Hashizume O, Yamazaki D, Miki H, Developmental cell, 2020.09, Papers

  • PRL3 pseudophosphatase activity is necessary and sufficient to promote metastatic growth., Kozlov G, Funato Y, Chen YS, Zhang Z, Illes K, Miki H, Gehring K, The Journal of biological chemistry,295(33) 11682-11692, 2020.08, Papers

  • Stress-responsive MTK1 SAPKKK serves as a redox sensor that mediates delayed and sustained activation of SAPKs by oxidative stress., Matsushita M, Nakamura T, Moriizumi H, Miki H, Takekawa M, Science advances,6(26) eaay9778, 2020.06, Papers

  • p18/Lamtor1-mTORC1 Signaling Controls Development of Mucin-producing Goblet Cells in the Intestine., Ito S, Nada S, Yamazaki D, Kimura T, Kajiwara K, Miki H, Okada M, Cell structure and function,45(2) 93-105, 2020.04, Papers

  • Excessive Mg<sup>2+</sup> Impairs Intestinal Homeostasis by Enhanced Production of ATP and Reactive Oxygen Species., Hashizume O, Funato Y, Yamazaki D, Miki H, Antioxidants & redox signaling, 2020.03, Papers

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